I think you’re correct, but I’m not sure how this translates to low carb dieting. You can make any healthy person eat too much if you give them insulin.
Gary Taubes argues that someone who eats more carbs is going to produce more insulin.
Froom Good Calories, Bad Calories:
This alternative hypothesis of obesity constitutes three distinct propositions. First, as I’ve said, is the basic proposition that obesity is caused by a regulatory defect in fat metabolism, and so a defect in the distribution of energy rather than an imbalance of energy intake and expenditure. The second is that insulin plays a primary role in this fattening process, and the compensatory behaviors of hunger and lethargy. The third is that carbohydrates, and particularly refined carbohydrates—and perhaps the fructose content as well, and thus perhaps the amount of sugars consumed—are the prime suspects in the chronic elevation of insulin; hence, they are the ultimate cause of common obesity.
I think the hypothesis is plausible, and if it were true the amount of carbohydrates people tolerate before their insulin secretion went crazy probably would vary greatly. I’m not sure if it applies to fructose, since its cell uptake doesn’t seem to be regulated by insulin. I’m pretty much ready to accept this idea as one of the major causes of obesity, but not necessarily the most important.
Does he say anything about insulin resistance in relation to this idea?
Far as as I understand he see a lot of insulin resistance as the result of elevated levels of blood insulin. I think he makes that argument in more detail and I’m probably not the right person to recount all the details.
I personally don’t think that there’s something like a ultimate cause of common obesity and that there are probably a lot of different factors at work.
Gary Taubes argues that someone who eats more carbs is going to produce more insulin.
Froom Good Calories, Bad Calories:
I think the hypothesis is plausible, and if it were true the amount of carbohydrates people tolerate before their insulin secretion went crazy probably would vary greatly. I’m not sure if it applies to fructose, since its cell uptake doesn’t seem to be regulated by insulin. I’m pretty much ready to accept this idea as one of the major causes of obesity, but not necessarily the most important.
Does he say anything about insulin resistance in relation to this idea?
Far as as I understand he see a lot of insulin resistance as the result of elevated levels of blood insulin. I think he makes that argument in more detail and I’m probably not the right person to recount all the details.
I personally don’t think that there’s something like a ultimate cause of common obesity and that there are probably a lot of different factors at work.