If you undergo ketosis your humoural triglycerides will be lysed.
Other people have written much on undergoing ketosis for weight loss, if you’re interested; beware though that much of their weight loss comes from loss of glycogen and water stores. Keeping yourself hydrated mitigates the latter, but liver glycogen stores will be depleted. Absent readily available glycogen, your body will break down humoural trigylcerides as it is your only remaining source of energy; glycogen stored in your muscles is left untouched lest you exercise extreme physical exertion. I now speculate, but at this point your body has become accustomed to using fats as its main energy source. If you enter into any sort of fasted state, be it through a caloric deficit, intermittent fasting, sleeping, etcetera, your body will lipolyse adipose cells for energy; this must happen or you will die—whatever prevented this previously will have been circumvented. Actually, if verily your body is so stubborn it won’t touch your adipose tissue, first you’d lose your skeletal muscle—then you’d die.
These are the physiological properties—if they don’t apply to you, then whatever genetic mutation causes your body’s nonconformity is unknown and I’d venture to guess has other effects as well.
If anyone tries this, pay attention to how fruity your urine/breath smells. If it becomes an overpowering scent, your blood pH may be too high. Either stop or take measures to rebalance your pH.
A zero-carb diet for a couple of weeks did not produce any ketosis as measured by a ketosis stick.
The most likely explanation is that you were eating a large amount of excess protein, most of which turns into glucose before it turns into ATP, and this was supplying (or nearly supplying) your nerve cells’ energy needs and inhibiting ketosis.
Lack of ketosis supplies at least part of an explanation of what’s going on with your metabolism. A fast google doesn’t supply any information about why ketosis might not happen, but it seems like a topic worth researching.
If you have too many amino acids in your blood your body doesn’t need to undergo ketosis. The generally recommended ratio is 65% fat or higher, %30 protein or less, and %5 carbohydrates or less.
Of course.
Sorry, I just realized we had a point of confusion due to a lack of clarity in my expression. I hope my edits are clearer.
If you undergo ketosis your fat cells will be lysed. Anecdotal reports aside, physiologically that must happen unless you are incapable of it (a genetic mutation which would—speculatively—have far-ranging effects).
Why must that happen? Ketosis means that lipolysis is occuring. Lipolysis does not inherently require that fat cells must be lysed as a physiological inevitability. ie. The fat that is to be broken down can itself come from diet instead of the lysing of fat cells.
You’re right—I wasn’t expressing myself thoroughly. I had the latent assumption of a caloric deficit, which as Eliezer has stated might not be enough to trigger breakdown of fat cells in some people. If the default fasted state of those people does not effect adequate catabolisation of their fat stores, then perhaps altering their body’s typical means of energy production would trigger lipolysis not just of humoural triglycerides but adipose cells as well when in a fasted state—exempli gratia through a caloric deficit, intermittent fasting, etcetera.
If you undergo ketosis your humoural triglycerides will be lysed.
Other people have written much on undergoing ketosis for weight loss, if you’re interested; beware though that much of their weight loss comes from loss of glycogen and water stores. Keeping yourself hydrated mitigates the latter, but liver glycogen stores will be depleted. Absent readily available glycogen, your body will break down humoural trigylcerides as it is your only remaining source of energy; glycogen stored in your muscles is left untouched lest you exercise extreme physical exertion. I now speculate, but at this point your body has become accustomed to using fats as its main energy source. If you enter into any sort of fasted state, be it through a caloric deficit, intermittent fasting, sleeping, etcetera, your body will lipolyse adipose cells for energy; this must happen or you will die—whatever prevented this previously will have been circumvented. Actually, if verily your body is so stubborn it won’t touch your adipose tissue, first you’d lose your skeletal muscle—then you’d die.
These are the physiological properties—if they don’t apply to you, then whatever genetic mutation causes your body’s nonconformity is unknown and I’d venture to guess has other effects as well.
If anyone tries this, pay attention to how fruity your urine/breath smells. If it becomes an overpowering scent, your blood pH may be too high. Either stop or take measures to rebalance your pH.
A zero-carb diet for a couple of weeks did not produce any ketosis as measured by a ketosis stick. Also lipolysis != dead fat cells.
The most likely explanation is that you were eating a large amount of excess protein, most of which turns into glucose before it turns into ATP, and this was supplying (or nearly supplying) your nerve cells’ energy needs and inhibiting ketosis.
Lack of ketosis supplies at least part of an explanation of what’s going on with your metabolism. A fast google doesn’t supply any information about why ketosis might not happen, but it seems like a topic worth researching.
Of course.
If you have too many amino acids in your blood your body doesn’t need to undergo ketosis. The generally recommended ratio is 65% fat or higher, %30 protein or less, and %5 carbohydrates or less.
Sorry, I just realized we had a point of confusion due to a lack of clarity in my expression. I hope my edits are clearer.
Why must that happen? Ketosis means that lipolysis is occuring. Lipolysis does not inherently require that fat cells must be lysed as a physiological inevitability. ie. The fat that is to be broken down can itself come from diet instead of the lysing of fat cells.
You’re right—I wasn’t expressing myself thoroughly. I had the latent assumption of a caloric deficit, which as Eliezer has stated might not be enough to trigger breakdown of fat cells in some people. If the default fasted state of those people does not effect adequate catabolisation of their fat stores, then perhaps altering their body’s typical means of energy production would trigger lipolysis not just of humoural triglycerides but adipose cells as well when in a fasted state—exempli gratia through a caloric deficit, intermittent fasting, etcetera.