I read Yvain’s report and compared it’s reasoning on potassium to the DRI DIETARY REFERENCE INTAKES FOR Water, Potassium, Sodium, Chloride, and Sulfate from Standing Committee on the Scientific Evaluation of Dietary
Reference Intakes of the Food and Nutrition Board (http://www.nal.usda.gov/fnic/DRI/DRI_Water/water_full_report.pdf). My understanding of potential concerns would be:
Serum potassium (responsible for the sodium-potassium pump etc.) is only very weakly linked to potassium intake, as the kidneys are very good at both excreting extra potassium and keeping potassium in the body if intake is low (though the kidneys are not as good at the latter as the former).
Potassium’s effect on lowering blood pressure is through the sodium-potassium balance, so potassium helps you mostly if your intake of sodium is too high.
Increased potassium intake’s potential beneficial effects are supposed come from a better acid/base balance in the body; but there is little experimental evidence (as opposed to epidemiological evidence)
I don’t think the epidemiological studies give reliable evidence for potassium, as the main source of dietary potassium are fresh vegetables, which already correlate with lower mortality on their own, as well as correlating with many lifestyle differences.
In animal studies the ‘good’ potassium was potassium bicarbonate, not potassium chloride, but most supplements have potassium chloride.
From other sources/common knowledge:
Sources of base (alkaline) ions should be buffered so that they are not released to the stomach, where they would serve to reduce the necessary acidity of the stomach fluids (very bad for your digestion) instead of helping your body’s acid/base balance.
My conclusion: it might indeed be beneficial to increase my potassium intake, but I would have to do it by increasing consumption of vegetables high in potassium rather than by taking supplements.
I read Yvain’s report and compared it’s reasoning on potassium to the DRI DIETARY REFERENCE INTAKES FOR Water, Potassium, Sodium, Chloride, and Sulfate from Standing Committee on the Scientific Evaluation of Dietary Reference Intakes of the Food and Nutrition Board (http://www.nal.usda.gov/fnic/DRI/DRI_Water/water_full_report.pdf). My understanding of potential concerns would be:
Serum potassium (responsible for the sodium-potassium pump etc.) is only very weakly linked to potassium intake, as the kidneys are very good at both excreting extra potassium and keeping potassium in the body if intake is low (though the kidneys are not as good at the latter as the former).
Potassium’s effect on lowering blood pressure is through the sodium-potassium balance, so potassium helps you mostly if your intake of sodium is too high.
Increased potassium intake’s potential beneficial effects are supposed come from a better acid/base balance in the body; but there is little experimental evidence (as opposed to epidemiological evidence)
I don’t think the epidemiological studies give reliable evidence for potassium, as the main source of dietary potassium are fresh vegetables, which already correlate with lower mortality on their own, as well as correlating with many lifestyle differences.
In animal studies the ‘good’ potassium was potassium bicarbonate, not potassium chloride, but most supplements have potassium chloride.
From other sources/common knowledge:
Sources of base (alkaline) ions should be buffered so that they are not released to the stomach, where they would serve to reduce the necessary acidity of the stomach fluids (very bad for your digestion) instead of helping your body’s acid/base balance.
My conclusion: it might indeed be beneficial to increase my potassium intake, but I would have to do it by increasing consumption of vegetables high in potassium rather than by taking supplements.