Basically, you don’t understand enough biology to see the difference between the two.
Viruses are evolved to interact with relatively stable targets. If we take COVID-19 for example, the ACE2 receptor is used by the virus. If all human cells would stop producing ACE2 receptors the infection doesn’t work.
There are mechanisms to present peptides or protein fragments on the cell wall. Those can break down, but that’s detectable by the immune system because the cell wall look differently.
If they are not broken down they will display any protein fragments that float around in the cell. It’s the nature of cancer that a lot of what floats around within a cell has mutations.
Not having a bunch of random mutations is the one thing that a cancer cell can’t do due to evolutionary pressure.
Basically, you don’t understand enough biology to see the difference between the two.
Viruses are evolved to interact with relatively stable targets. If we take COVID-19 for example, the ACE2 receptor is used by the virus. If all human cells would stop producing ACE2 receptors the infection doesn’t work.
There are mechanisms to present peptides or protein fragments on the cell wall. Those can break down, but that’s detectable by the immune system because the cell wall look differently.
If they are not broken down they will display any protein fragments that float around in the cell. It’s the nature of cancer that a lot of what floats around within a cell has mutations.
Not having a bunch of random mutations is the one thing that a cancer cell can’t do due to evolutionary pressure.
The lipid nanoparticle mechanism used by the mRNA vaccines could have had a cancer destroying payload.
As I understand it the lipids merge with cell membranes, it’s receptor independent.
So unless the cancer cell can evolve to not have a cell membrane it’s vulnerable.