Thank you, Carl Shulman, for correcting my misinformation. It’s difficult for one to know which references are reliable, when one is not in the field.
@Carl Shulman: The largest hypothesized effects of the disease alleles would be only a small fraction of the Ashkenazim advantage: they just aren’t frequent enough.
Dr. Bostrom cites this paper (so I considered it might be reliable) in his treatise on cognitive enhancement: “Natural History of Ashkenazi Intelligence” by Gregory Cochran, Jason Hardy, Henry Harpending. Speaking of the incidence of of the Ashkenazim mutations, they state: “the probability of having at least one allele from these disorders is 59%.”. As I understand it, these disorders are exceedingly rare in non-Ashkenazim. Are these authors simply incorrect, or did you mean that a 59% incidence just isn’t frequent enough? That incidence is very close to the intelligence distribution I read between mutated and non-mutated Ashkenazim. Coincidence? Or already discredited?
Thank you, Carl Shulman, for correcting my misinformation. It’s difficult for one to know which references are reliable, when one is not in the field.
@Carl Shulman: The largest hypothesized effects of the disease alleles would be only a small fraction of the Ashkenazim advantage: they just aren’t frequent enough.
Dr. Bostrom cites this paper (so I considered it might be reliable) in his treatise on cognitive enhancement: “Natural History of Ashkenazi Intelligence” by Gregory Cochran, Jason Hardy, Henry Harpending. Speaking of the incidence of of the Ashkenazim mutations, they state: “the probability of having at least one allele from these disorders is 59%.”. As I understand it, these disorders are exceedingly rare in non-Ashkenazim. Are these authors simply incorrect, or did you mean that a 59% incidence just isn’t frequent enough? That incidence is very close to the intelligence distribution I read between mutated and non-mutated Ashkenazim. Coincidence? Or already discredited?