I mean how a bacterium will make lactase when there’s lactose, but won’t make lactose when there isn’t lactose.
It’s quite easy to have a receptor protein that binds to lactose and then leads to a protein being expressed that turns lactose into lactase.
“amount of available food in the environment” is not as simple to measure inside of a cell and as a result, the regulation is much more complex.
I’m asking why orexin wouldn’t be physiologically adaptive to the amount of food that’s generally around.
That assumes that orexin is independent of food that’s around, which clearly isn’t true. Fasting increases orexin levels, it’s just that chronic food restriction that doesn’t change it.
Hm. So orexin is increased initially during a fast, and also when eating high-caloric food? That’s weird. Do people who eat high-caloric food need less sleep?
It’s quite easy to have a receptor protein that binds to lactose and then leads to a protein being expressed that turns lactose into lactase.
“amount of available food in the environment” is not as simple to measure inside of a cell and as a result, the regulation is much more complex.
That assumes that orexin is independent of food that’s around, which clearly isn’t true. Fasting increases orexin levels, it’s just that chronic food restriction that doesn’t change it.
Hm. So orexin is increased initially during a fast, and also when eating high-caloric food? That’s weird. Do people who eat high-caloric food need less sleep?
Where did you get the claim about eating high caloric food?
“Here, we review a fat-burning mechanism that is turned on by the brain hormone orexin during high-caloric food consumption.”
Maybe they’re not saying that the diet upregulates orexin?