Wouldn’t it expand the size of the genome and potentially affect the distance between promoters/enhancers and target genes, causing a loss in a cell’s ability to appropriately regulate translation in response to perturbation?
To some extent, though presumably the vast majority of copies will be into non-functional sequence, and copies into functional sequence will often result in a defective cell which is quickly removed. The expansion of the genome size shouldn’t be significant until the count is already way out of control; a transposon is tiny compared to the whole genome.
To some extent, though presumably the vast majority of copies will be into non-functional sequence, and copies into functional sequence will often result in a defective cell which is quickly removed. The expansion of the genome size shouldn’t be significant until the count is already way out of control; a transposon is tiny compared to the whole genome.