I was thinking of areas along the gum-tooth interface having a local environment that normally promote tooth demineralization and cavities. After Lumina, that area could have high chronic acetaldehyde levels. In addition, the adaption of oral flora to the chronic presence of alcohol could increase first-pass metabolism, which increases acetaldehyde levels locally and globally during/after drinking.
I don’t know how much Lumina changes the general oral environment, but I think you might be able to test this by seeing how much sugar you can put in your mouth before someone else can smell the fruity scent of acetaldehyde on your breath? I’m sure someone else can come up with a better experiment.
I was thinking of areas along the gum-tooth interface having a local environment that normally promote tooth demineralization and cavities. After Lumina, that area could have high chronic acetaldehyde levels. In addition, the adaption of oral flora to the chronic presence of alcohol could increase first-pass metabolism, which increases acetaldehyde levels locally and globally during/after drinking.
I don’t know how much Lumina changes the general oral environment, but I think you might be able to test this by seeing how much sugar you can put in your mouth before someone else can smell the fruity scent of acetaldehyde on your breath? I’m sure someone else can come up with a better experiment.