I’ve heard an argument that Mendel was actually counter-productive to the development of genetics. That if you go and actually study peas like he did, you’ll find they don’t make perfect Punnett squares, and from the deviations you can derive recombination effects. The claim is he fudged his data a little in order to make it nicer, then this held back others from figuring out the topological structure of genotypes.
I’ve heard, in this context, the partial counterargument that he was using traits which are a little fuzzy around the edges (where is the boundary between round and wrinkled?) and that he didn’t have to intentionally fudge his data in order to get results that were too good, just be not completely objective in how he was determining them.
Of course, this sort of thing is why we have double-blind tests in modern times.
I’ve heard an argument that Mendel was actually counter-productive to the development of genetics. That if you go and actually study peas like he did, you’ll find they don’t make perfect Punnett squares, and from the deviations you can derive recombination effects. The claim is he fudged his data a little in order to make it nicer, then this held back others from figuring out the topological structure of genotypes.
I’ve heard, in this context, the partial counterargument that he was using traits which are a little fuzzy around the edges (where is the boundary between round and wrinkled?) and that he didn’t have to intentionally fudge his data in order to get results that were too good, just be not completely objective in how he was determining them.
Of course, this sort of thing is why we have double-blind tests in modern times.