Evidence in mice studies does indicate that earlier therapies (for example of senolytics) do facilitate greater life extension. However, with better anti-aging technologies the ‘switch’ (from the paper you refer to) could theoretically be reversed, as there’s no biological law that would prevent restoring a phenotypically older individual back to a more youthful state.
Evidence in mice studies does indicate that earlier therapies (for example of senolytics) do facilitate greater life extension. However, with better anti-aging technologies the ‘switch’ (from the paper you refer to) could theoretically be reversed, as there’s no biological law that would prevent restoring a phenotypically older individual back to a more youthful state.