I mostly avoid this kind of analysis as food and digestion is an extremely complicated subject.
Glycemic load, for instance, is known to behave in a quite non-linear way. If you mix two foods, the total glycemic load might be very different from sum of the individual parts. It also depends on a lot of other factors, like person-specific factors and situation-specific factors.
[This] suggests that in parallel with the bacterial effects of sugars on dental and periodontal health, acellular flours, sugars, and processed foods produce an inflammatory microbiota via the upper gastrointestinal tract, with fat able to effect a “double hit” by increasing systemic absorption of lipopolysaccharide. This model is consistent with a broad spectrum of reported dietary phenomena.
I mostly avoid this kind of analysis as food and digestion is an extremely complicated subject.
Glycemic load, for instance, is known to behave in a quite non-linear way. If you mix two foods, the total glycemic load might be very different from sum of the individual parts. It also depends on a lot of other factors, like person-specific factors and situation-specific factors.
More about problems with glycemic load
Interesting. One referenced study (Comparison with ancestral diets) gives an interesting interpreatation: