One update to make is that the dietary absorption figure (.78%) used by Mitkus to map from the dietary MRL to intravenous seems to be off by a factor of 8 (the ATSDR says average dietary bioavailability is 0.1%; the .78% number is out of range from all other study estimates and doesn’t even make clear sense as a takeaway from the Al-26 study that it came from); so the exposure amount from vaccines appears to be basically equal to the MRL, rather than well below.
So that would put us at exposure comparable to 1% of what’s needed for “observable effects” in mice according to the study cited by the ATDSR;
A second possible update: if you look harder for those observable neurological effects, apparently you can find them in rodents at a tenth of that level?
”there are numerous reports of neurotoxic effects in mice and rats, confirmed by coherent neurobiological alterations, for oral doses of Al much < 26 mg/kg/d: 6 mg/kg/d reported in 1993 [86], 5.6 mg/kg/ d reported in 2008 and 2009 [87,88], 10 mg/kg/d reported in 2016 [89], 3.4 mg/kg/d reported in 2016 and 2017 [90,91], and even 1.5 mg/kg/d reported in 2017 [92].”
I don’t know how to assess the reliability of this literature.
Third possible issue to litigate is whether the specific form of aluminum matters, and the specific form in vaccines can pass the BBB more easily via immune cells. Comment chain for that issue here
related old blog post I wrote
Great post!
One update to make is that the dietary absorption figure (.78%) used by Mitkus to map from the dietary MRL to intravenous seems to be off by a factor of 8 (the ATSDR says average dietary bioavailability is 0.1%; the .78% number is out of range from all other study estimates and doesn’t even make clear sense as a takeaway from the Al-26 study that it came from); so the exposure amount from vaccines appears to be basically equal to the MRL, rather than well below.
So that would put us at exposure comparable to 1% of what’s needed for “observable effects” in mice according to the study cited by the ATDSR;
A second possible update: if you look harder for those observable neurological effects, apparently you can find them in rodents at a tenth of that level?
This source says:
”there are numerous reports of neurotoxic effects in mice and rats, confirmed by coherent neurobiological alterations, for oral doses of Al much < 26 mg/kg/d: 6 mg/kg/d reported in 1993 [86], 5.6 mg/kg/ d reported in 2008 and 2009 [87,88], 10 mg/kg/d reported in 2016 [89], 3.4 mg/kg/d reported in 2016 and 2017 [90,91], and even 1.5 mg/kg/d reported in 2017 [92].”
I don’t know how to assess the reliability of this literature.
Third possible issue to litigate is whether the specific form of aluminum matters, and the specific form in vaccines can pass the BBB more easily via immune cells. Comment chain for that issue here
It seems that mRNA vaccines basically don’t need any adjuvants because the RNA in the vaccine is already working as an adjuvant.
That suggests that any RNA/DNA would work and you don’t need the specific DNA of the host. Giving some random DNA might work as an adjuvant.
DNA also has the advantage that it’s a big molecule and thus won’t cross the blood-brain barrier.