What asymmetries did you introduce into your simulations that lead to a difference? Models with no gender differences but with mandatory sexual reproduction usually tend to be 50⁄50 in my experience.
My models had humans with their full 46 chromosomes and multiple genes per chromosome. In addition, I have transposons on those chromosomes. I also tried to have a model of mating behavior where males and females obviously have different roles.
Mutations on the x-chromosome lead more frequently to pregnancy termination in male offspring. This is pretty obvious given that female offspring have more redundancy when it comes to the X chromosome.
The transposon-related pregnancy terminations that in turn terminate more female pregnancies than male ones are less obvious. I think I have some insight there that could be publishable. If anyone wants to collaborate on a paper I’m happy to say more privately.
Transposons and their effects get often not taken as seriously as they should.
What asymmetries did you introduce into your simulations that lead to a difference? Models with no gender differences but with mandatory sexual reproduction usually tend to be 50⁄50 in my experience.
My models had humans with their full 46 chromosomes and multiple genes per chromosome. In addition, I have transposons on those chromosomes. I also tried to have a model of mating behavior where males and females obviously have different roles.
Mutations on the x-chromosome lead more frequently to pregnancy termination in male offspring. This is pretty obvious given that female offspring have more redundancy when it comes to the X chromosome.
The transposon-related pregnancy terminations that in turn terminate more female pregnancies than male ones are less obvious. I think I have some insight there that could be publishable. If anyone wants to collaborate on a paper I’m happy to say more privately.
Transposons and their effects get often not taken as seriously as they should.